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DNA damage promotes mutations of suppressor in tumor such as calcium/calmodulin dependent serine protein kinase (CASK), p53, as well as stimulation of the epidermal growth factor receptor (EFGR) signaling pathway, which are important early events in gastric carcinogenesis (86, 87)

NFT pretreatment resulted in a marked shift of LD 50 toward higher concentrations of either NFT or paraquat required for the induction of cell damage compared to cells without pretreatment

The high astrocyte specificity of MAO-B around dense-core A plaques enables more precise interrogation of astrocyte-related pathophysiology in the living brain in the context of neurodegenerative disease (Jaisa-Aad et al., 2024)