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The major risk factors established for severe COVID-19 infection and relative glutathione deficiency found in COVID-19 infected patients with moderate-to-severe illness have converged me to two very important conclusions: (1) oxidative stress contributes to hyper-inflammation of the lung leading to adverse disease outcomes such as acute respiratory distress syndrome, multiorgan failure and death

For NPs with similar chemical compositions, different surface modifications lead to toxic effects in varying degrees (Zhang et al., 2021)

Lipid peroxide 4-hydroxynonenal (4HNE) resulting from ferroptosis can also promote NF-B activation, thus inducing inflammatory mediator response [29]