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Here, we propose that disrupting the ferroptosis defense system, particularly the GST family, represents a potential therapeutic strategy for EMs

ScharfsteinJSKeaneyJFJr.SlivkaAWelchGNVitaJAStamlerJSet alIn vivo transfer of nitric oxide between a plasma protein-bound reservoir and low-molecular weight thiols

have shown that inhibition of PSC activation is associated with a decrease in islet fibrosis and an increase in insulin secretion [35], indicating the potential role of PSC activation in -cell failure